Antibiotics

After pitiful a lot of devastating epidemic attacks and mass deaths, humanity invented antibiotics and started applying them against cognize bacterial diseases. Using antibiotics for the last 6 decades, we are now confront the results of the bear upon caused by genetic changes and mutations in bacteria. This merchant ship be explained by one of the basic principles of evolution natural selection, which suggests that the fittest and the strongest h white-haired water, and the weakest disappear.Bacteria are acellular organisms, which are very vulnerable to mutations. It is known that the most of the mutations affect the organisms. alone certainly, there are some chances of positive outcomes of mutations as well. Mitosis of bacteria is a very fast process, which brings to rapid growth of the population. Having such huge issue forth of bacteria, the chances of positive mutation are higher, therefore, much and more bacteria can become resistant to antibiotics treatment.Nowadays, different types of pathogen bacteria can already survive the existing antibiotics, and using an excessive amount of antibiotics we dramatically fastened the process of natural selection in bacteria evolution. This situation threatens the effectiveness of conventional treatment methods to bacterial diseases. That is why world scientists are facing the prerequisite of searching for new antibiotics, either modifying the known ones or looking for something perfectly different.According to the research of the Harvard School of Public Health, in 2005 ..more than 40% of strep pneumoniae strains in the United States could resist both penicillin and erythromycin (Powledge, 2004). The other researches, ground on mathematical modeling, show that in the nearest future more and more species of bacteria will be able to resist old antibiotics. Therefore, if no new antibiotics are available, well become totally unprotected against backbreaking diseases, like tuberculosis, etc.BibliographyPowl edge, T. M. (2004, February 17). New AntibioticsResistance Is Futile. PLoS Biol 2(2) e53